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GHK-Cu activates genes encoding collagen I, III, and elastin production while simultaneously downregulating matrix metalloproteinases (MMPs) the enzymes responsible for degrading periodontal ligament fibers in periodontitis

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Their mechanisms diverge on several key fronts: GHK-Cu has documented antioxidant effects and MMP/TIMP modulatory activity, while retinoids primarily drive epidermal turnover and reduce UV-induced MMP activation

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